Crossbreeding with TASK-1- and TASK-3-deficient mice will allow the investigation of specific channel function in interneurons.
Possible pathophysiological alterations of K2P channels will be analyzed in a mouse model of human absence epilepsy (backcross of C3H/HeJ mice).
Staff:
for1086.uni-muenster.deDas Einkreuzen von TASK-1- und TASK-3-KO-Mäusen, erlaubt die Analyse der spezifischen Kanalfunktion in Interneuronen.
Pathophysiologische Veränderungen der Expression von K2P-Kanälen sollen in einem Mausmodell der menschlichen Absence-Epilepsie (Rückkreuzungen von C3H/HeJ-Mäusen) analysiert werden.
Mitarbeiter:
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